Insulin and glucose homeostasis in childhood cancer survivors treated with abdominal radiation: A pilot study
Academic Article
Overview
MeSH Major
Antibody Affinity
Leukemia
Peptide Fragments
Receptors, Antigen, T-Cell
Single-Chain Antibodies
WT1 Proteins
abstract
This study suggests that radiation-induced damage to the insulin-producing β-cells is an unlikely explanation for the early derangements in glucose metabolism observed after abdRT. Research into alternative pathways leading to diabetes after abdRT is needed.
