Global reimbursement: The authors reply Letter uri icon

Overview

MeSH Major

  • Critical Illness
  • Intensive Care Units
  • Models, Statistical
  • Patient Discharge

abstract

  • These results indicate that the observed decrease in CBF during dipyridamole stress was caused by a decrease in PaCO(2) rather than by any direct action of dipyridamole on CBF. The decrease in PaCO(2) during dipyridamole stress was most likely due to hyperventilation, which was a side effect of adenosine. These results support the hypothesis that circulating adenosine is largely prevented from binding to adenosine receptors of cerebral vessels by the blood-brain barrier.

publication date

  • August 2011

Research

keywords

  • Letter

Identity

Digital Object Identifier (DOI)

  • 10.1377/hlthaff.2011.0767

Additional Document Info

start page

  • 1616

end page

  • 20

volume

  • 30

number

  • 8