Asymmetric Dimethyl l-Arginine (ADMA) is a critical regulator of myocardial reperfusion injury Academic Article uri icon

Overview

MeSH Major

  • Arginine
  • Endothelium, Vascular
  • Myocardial Reperfusion Injury
  • Nitric Oxide
  • Nitric Oxide Synthase

abstract

  • Accumulation of tissue ADMA by impairment of DDAH was found to be a significant determinant of reperfusion injury. Our results indicate that ADMA could be a potential new target for the treatment of myocardial ischemia/reperfusion injury.

publication date

  • July 15, 2007

Research

keywords

  • Academic Article

Identity

Language

  • eng

Digital Object Identifier (DOI)

  • 10.1016/j.cardiores.2007.04.030

PubMed ID

  • 17559823

Additional Document Info

start page

  • 417

end page

  • 25

volume

  • 75

number

  • 2