Azoospermia in patients heterozygous for a mutation in SYCP3 Academic Article uri icon

Overview

MeSH Major

  • Mutation
  • Nuclear Proteins
  • Oligospermia

abstract

  • We suggest that SYCP3 has an essential meiotic function in human spermatogenesis that is compromised by the mutant protein via dominant negative interference.

publication date

  • November 22, 2003

Research

keywords

  • Academic Article

Identity

Language

  • eng

Digital Object Identifier (DOI)

  • 10.1016/S0140-6736(03)14845-3

PubMed ID

  • 14643120

Additional Document Info

start page

  • 1714

end page

  • 9

volume

  • 362

number

  • 9397