Endothelial dysfunction induced by hyperhomocyst(e)inemia: Role of asymmetric dimethylarginine Academic Article uri icon

Overview

MeSH Major

  • Arginine
  • Endothelium, Vascular
  • Hyperhomocysteinemia

abstract

  • Our results suggest that experimental hyperhomocyst(e)inemia leads to accumulation of the endogenous NO synthase inhibitor ADMA, accompanied by varying degrees of endothelial dysfunction according to the preexisting state of cardiovascular health.

publication date

  • August 26, 2003

Research

keywords

  • Academic Article

Identity

Language

  • eng

Digital Object Identifier (DOI)

  • 10.1161/01.CIR.0000085067.55901.89

PubMed ID

  • 12912818

Additional Document Info

start page

  • 933

end page

  • 8

volume

  • 108

number

  • 8