RECURRENT PULMONARY OEDEMA IN HYPERTENSION DUE TO BILATERAL RENAL ARTERY STENOSIS: TREATMENT BY ANGIOPLASTY OR SURGICAL REVASCULARISATION Academic Article Article uri icon

Overview

MeSH Major

  • Piperazines
  • Poxviridae
  • Protein Kinase Inhibitors
  • Proto-Oncogene Proteins c-abl
  • Pyrimidines

abstract

  • 11 patients with atheromatous renovascular hypertension had a history of multiple episodes of pulmonary oedema. 7 had stenosis of both renal arteries, 2 had stenosis of the artery to a solitary kidney, and 2 had unilateral stenosis with an intact contralateral kidney. Successful revascularisation (by angioplasty in 8, and surgery in 3) improved blood pressure and renal function, and virtually eliminated pulmonary oedema. In a second series of 55 consecutive patients with azotaemia and renovascular hypertension, pulmonary oedema occurred in 13 (23%). Blood pressure and renal function were not significant predictors of pulmonary oedema, but coronary heart disease and bilateral (vs unilateral) renal artery stenosis were. Bilateral renal artery stenosis may be a specific and treatable predisposing factor to pulmonary oedema in azotaemic hypertensive patients.

publication date

  • September 3, 1988

Research

keywords

  • Academic Article

Identity

Digital Object Identifier (DOI)

  • 10.1016/S0140-6736(88)92668-2

PubMed ID

  • 2900930

Additional Document Info

start page

  • 551

end page

  • 2

volume

  • 332

number

  • 8610