- The oxygen free radicals originating from mitochondrial complex I contribute to oxidative brain injury following hypoxia-ischemia in neonatal mice. The Journal of neuroscience : the official journal of the Society for Neuroscience. 2012 Academic Article GET IT
- Mild hypoxemia during initial reperfusion alleviates the severity of secondary energy failure and protects brain in neonatal mice with hypoxic-ischemic injury. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. 2012 Academic Article GET IT
- Complement component c1q mediates mitochondria-driven oxidative stress in neonatal hypoxic-ischemic brain injury. The Journal of neuroscience : the official journal of the Society for Neuroscience. 2010 Academic Article GET IT
Complement component C3 mediates inflammatory injury following focal cerebral ischemia.
Times cited: 107